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Hyperbaric Oxygenation: Mitochondrial Activity and Brain Physiological Functions Chapter 5 Effect of the Pressure Level on the Oxygen Toxicity Process This research investigates how various levels of high-pressure oxygen impact the metabolic, haemodynamic, and electrical functions of the living rat brain to better understand the mechanisms of oxygen toxicity. By monitoring real-time changes in brain signals, the study identifies that while toxicity generally intensifies as pressure increases, the 60-psi mark serves as a critical turning point where the biological kinetics of the process shift significantly. The data reveal a highly synchronised sequence of events—beginning with vascular changes (vasodilation), followed by tonic-clonic convulsions, and ending with metabolic cycles—suggesting that the primary cause of toxicity may be related to cellular membrane instability and ion imbalances rather than direct mitochondrial failure. Ultimately, the text provides a structured correlation between environmental pressure and the physiological onset of seizures, establishing a predictable timeline for the progression of hyperbaric oxygen poisoning.