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Bootleg spirits, dodgy “house pours,” and travel cocktails gone wrong—methanol isn’t just a bad hangover; it’s a toxic alcohol that can cost vision and life. In this video, we break down methanol poisoning in plain English with an ED lens, using real-world Australian context (think Bali arak cases and festival mixes). What you’ll learn: The pathway from methanol → formic acid → optic nerve injury and severe metabolic acidosis. Red flags: headache, vomiting, abdominal pain, blurred/“snowfield” vision, photophobia, and Kussmaul respirations. Lab clues: rising anion gap metabolic acidosis, early ↑ osmol gap (may normalise later), lactate “look-alikes.” Immediate actions: ABCs, antidotes (fomepizole or ethanol if fomepizole unavailable), bicarbonate for acidosis, folinic/folic acid to enhance formate metabolism. When to dialyse: significant acidosis, visual symptoms, end-organ toxicity, high methanol level, or deterioration despite antidote/support. Key differentials vs ethylene glycol and isopropanol, plus practical ED tips for rural/remote transfers in Australia. This is educational content, not medical advice. If you suspect poisoning, call 000 (Australia) or present to an ED immediately. If you’re prepping for ACEM, stick around for the quick decision frameworks. If you’re a traveller, this could save your eyesight. Subscribe for more emergency medicine content, and drop your questions below.