Perimembranous ventricular Septal Defect with Severe Tricuspid Regurgitation скачать в хорошем качестве

Perimembranous ventricular Septal Defect with Severe Tricuspid Regurgitation

Discussion: Mechanisms of tricuspid regurgitation in patient with perimembranous VSD: 1) Tricuspid valves clefts or dysplasia. 2) VSD jet pushing the tricuspid anterior leaflet to open tricuspid valves orifice. Mechanisms of spontaneous closure of membranous VSD: The tricuspid valve was responsible for most common mechanism of closure(85%) by either reduplication of leaflet tissue or adherence of septal leaflet to the margins of defect. Membranous VSD diminish in size or close spontaneously by aneurysm formation. So-call ventricular septal aneurysm is not a true aneurysm of the septaum membranaceum but rather a tissue out pouching from the tricuspid valve. Refrences: 1) A. Rebecca Snider, MD et al: Echocardiography in pediatric Heart Disease,2nd edi 1997,Mosby-Year Book Inc.2) DonaldJ. Hagler, MD et al: Mechanism of tricuspid regurgitatio in paramembranous VSD, JASE,15:364,2002.